Thanks for your post oldcorollas but SRT501 is a drug it isn't resveratrol. GlaxoSmithKline are trying to make a synthetic version of resveratrol which can activate the SIRT1 activators which is believed to be responsible for "life extension".
Basically resveratrol does do this but it is very weak so GlaxoSmithKline are trying to make a synthetic much more powerful drug which does the same thing. They are basing their drug on the research that has been done with resveratrol.
Extra virgin olive oil will increase your HDL cholesterol this is why when ghosty does his next "BEFORE" set of blood tests he will be using the EVOO from now until then.
And then when he is using the res100 he will still be doing the exact same thing so that all variables are the same.
When you read what you have quoted from Linus Pauling Institute it clearly states the following: "In estrogen receptor-positive breast cancer cells, resveratrol acted as an estrogen agonist
in the absence of the endogenous estrogen"
So when natural estrogen is not present it will act as an estrogen.
"17beta-estradiol, but acted as an estrogen antagonist in the presence of 17beta-estradiol "
and when estrogen is present it will act as an anti estrogen.
For those who dont understand what an agonist and antagonist is:
An agonist is a chemical that binds to a
receptor of a cell and triggers a response by that cell. Agonists often mimic the action of a naturally occurring substance. Whereas an agonist causes an action, an
antagonist blocks the action of the agonist and an
inverse agonist causes an action opposite to that of the agonist.
but there is a lot of hype...
