The only objective measure of progress over time is weight on the bar and/or gains. Not soreness post workout.
I see people around over the years saying that you have to get sore to get bigger. Which I somewhat dis-agree with as I've gain a fair amount of muscle with rarely getting sore.
I've been lifting for quite a few years now progressively adding more and more volume (and weight when appropriate) but still seldom get sore muscles.
It's got to the point where I don't know if I should keep adding more volume or not. A typical bench/tricep workout that I don't feel the next day...
Bench Press - 10sets x 12-15reps
Incline Bench Press - 10sets x 12-15reps
More Bench Press - 10sets x 12-15reps
Dumbbell Bench Press - 10sets x 15-20reps
Tricep Exercise 1 - 10sets x 15-20reps
Tricep Exercise 2 - 10sets x 15-20reps
All sets are as heavy as I can do for the desired rep range, if I can get extra reps, I do.
Most other muscles are trained with the same volume.
I change the exercises around every 3-4 months, but pretty limited as I train in my house.
So was wondering how much training other people need to do before getting sore?
60 sets at a challenging level of resistance? Either your strength endurance is phenomenal or the weight could be a lot more challenging. Have you tried 30 set workouts with heavier weight?
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I don't actually believe that muscle can or feel pain sense, I think where pain is coming from is the surrounding tissue like the bursar, ligaments and tendons, which would explain why pain is felt after an exercise you're not use to or had done in a while.
Skeletal muscle contains nociceptors which are responsible for the sensation of pain -
Muscle pain is produced by the activation of specific receptors (so-called nociceptors): these receptors are specialized for the detection of stimuli that are objectively capable of damaging tissue and that are subjectively perceived as painful. They consist of free nerve endings and are connected to the central nervous system (CNS) by way of unmyelinated (group IV) or thinly myelinated (group III) fibers. They can be sensitized and activated by strong mechanical stimuli, such as trauma or mechanical overloading, as well as by endogenous inflammatory mediators including bradykinin (BK), serotonin, and prostaglandin E2 (PGE2).
Two activating chemical substances are particularly important for the generation of muscle pain: adenosine triphosphate (ATP) and protons (H+ ions). These chemical irritants activate nerve endings by binding to receptor molecules located in the membrane of the nerve ending. ATP activates muscle nociceptors mainly by binding to the P2X3 receptor molecule, H+ mainly by binding to the receptor molecules TRPV1 (transient receptor potential vanilloid 1) and ASICs (acid-sensing ion channels) (4). These receptor molecules are channel proteins that span the membrane of the nerve ending and mainly permit Na+ ions to enter the neuron. These Na+ ions then induce neural excitation.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2696782/
what's it sayin?
I've said it before and @Fadi; touched on this: exercises with an exaggerated stretch component such as SLDL, calf raises, lying tricep ext tend to result in more sorrness than say squats or ohp where there is little muscle stretch. Happens to me anyway.
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