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staunch308
New member
Some people use it during PTC to help remain anabolic, same with GH. I wouldn't use it unless like 0ni said your maxing out on AAS, because of one finding a source that is legit and two finding the money lol and the gains won't make you that impressed
Skalatharx
Member
Oni for mod?
staunch308
New member
Sorry typo, PCT haha
0ni
Registered Rustler
I have a cyclist mate who used IGF and he didn't get much out of it tbh
What we used to do when coming off was low dose an oral like dianabol or oxandrolone at about 10mg a day, run PCT for 2 weeks like that so that your testosterone gets restarted again and you remain in an anabolic state while you're waiting for that. Dianabol is used because it won't shut you down totally, it suppresses you instead (they are two different things) and so it is possible to low dose it and run PCT until you're back to acceptable levels of testosterone, dropping the anabolics totally and running PCT a few weeks more as usual
Worked with cycling but we were not trying to hold as much mass and strength, it was more of a work capacity thing, although a quick google around has given some good feedback of bodybuilders who use 10-20mg of dianabol at the start of PCT for this reason
What we used to do when coming off was low dose an oral like dianabol or oxandrolone at about 10mg a day, run PCT for 2 weeks like that so that your testosterone gets restarted again and you remain in an anabolic state while you're waiting for that. Dianabol is used because it won't shut you down totally, it suppresses you instead (they are two different things) and so it is possible to low dose it and run PCT until you're back to acceptable levels of testosterone, dropping the anabolics totally and running PCT a few weeks more as usual
Worked with cycling but we were not trying to hold as much mass and strength, it was more of a work capacity thing, although a quick google around has given some good feedback of bodybuilders who use 10-20mg of dianabol at the start of PCT for this reason
staunch308
New member
0ni can you explain the difference between supressing and shut down? Always considered them the same, reduction of natural androgen production and atrophy of the nads?
0ni
Registered Rustler
"Shutdown", is defined by a complete inhibition of the Pituitary/Testes, resulting in a total cessation of endogenous androgen production.
some androgens will only suppress endogenous androgen production, resulting in a decreased testosterone level, but not a complete shutdown. (Turinabol, Anavar, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan)
Very Androgenic/Progestenic/Estrogenic steroids (Trenbolone, Nandrolone, Anadrol, Testosterone) cause a complete shutdown of endogenous hormone production.
The Hypothalamus has Androgen, Estrogen, and Progesterone receptors. Each and every anabolic steroid affects these receptors differently. Some steroids affect all receptors, while some only affect one type of receptor, while others have very little effect on any of these receptors.
Steroids that cause an oversaturation too many receptors activated) of these various hormone receptors, will cause shutdown.
Steroids that do not cause an oversaturation of any of these various hormone receptors, will not cause shutdown.
The Following drugs either directly or indirectly activate estrogen receptors, to varying degrees:
Testosterone
Methandrostenolone
Mathandriol
Oxymetholone
Nandrolone
Boldenone
The Following drugs either directly or indirectly activate progesterone receptors, to varying degrees:
Nandrolone
Trenbolone
Oxymetholone
And all AAS indirectly or directly activates androgen receptors (that's why we take them!!)
As we can see, the steroids that cause HPTA shutdown either oversaturate one specific receptor, or they activate too many total receptors(Androgen/Estrogen/Progesterone)
For instance, Trenbolone causes HPTA shutdown because it oversaturates both the androgen and the progesterone receptors.
Testosterone causes shutdown because it converts to estrogen and DHT, therefore, it oversaturates the Androgen/Estrogen receptors.
As we can also see, the steroids that don't cause shutdown of the HPTA, do not oversaturate ANY of the different hormone receptors, and thus, do not cause shutdown.
Methenolone(Primobolan) does not possess any Estrogenic or Progestational activity whatsoever. It does, by virtue of being an anabolic steroid, posses a small Androgenic component. Because it lacks any estrogenic/progestational component, and it lacks a strong Androgenic component, it will not cause shutdown!
Oxandrolone(Anavar) posseses no Estrogenic/Progestational component either. And, it also lacks a strong androgenic component. Thus, Anavar will not cause shutdown.
It must also be noted, that ANY steroid in large enough dosages for long enough durations, can cause shutdown of the HPTA
some androgens will only suppress endogenous androgen production, resulting in a decreased testosterone level, but not a complete shutdown. (Turinabol, Anavar, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan)
Very Androgenic/Progestenic/Estrogenic steroids (Trenbolone, Nandrolone, Anadrol, Testosterone) cause a complete shutdown of endogenous hormone production.
The Hypothalamus has Androgen, Estrogen, and Progesterone receptors. Each and every anabolic steroid affects these receptors differently. Some steroids affect all receptors, while some only affect one type of receptor, while others have very little effect on any of these receptors.
Steroids that cause an oversaturation too many receptors activated) of these various hormone receptors, will cause shutdown.
Steroids that do not cause an oversaturation of any of these various hormone receptors, will not cause shutdown.
The Following drugs either directly or indirectly activate estrogen receptors, to varying degrees:
Testosterone
Methandrostenolone
Mathandriol
Oxymetholone
Nandrolone
Boldenone
The Following drugs either directly or indirectly activate progesterone receptors, to varying degrees:
Nandrolone
Trenbolone
Oxymetholone
And all AAS indirectly or directly activates androgen receptors (that's why we take them!!)
As we can see, the steroids that cause HPTA shutdown either oversaturate one specific receptor, or they activate too many total receptors(Androgen/Estrogen/Progesterone)
For instance, Trenbolone causes HPTA shutdown because it oversaturates both the androgen and the progesterone receptors.
Testosterone causes shutdown because it converts to estrogen and DHT, therefore, it oversaturates the Androgen/Estrogen receptors.
As we can also see, the steroids that don't cause shutdown of the HPTA, do not oversaturate ANY of the different hormone receptors, and thus, do not cause shutdown.
Methenolone(Primobolan) does not possess any Estrogenic or Progestational activity whatsoever. It does, by virtue of being an anabolic steroid, posses a small Androgenic component. Because it lacks any estrogenic/progestational component, and it lacks a strong Androgenic component, it will not cause shutdown!
Oxandrolone(Anavar) posseses no Estrogenic/Progestational component either. And, it also lacks a strong androgenic component. Thus, Anavar will not cause shutdown.
It must also be noted, that ANY steroid in large enough dosages for long enough durations, can cause shutdown of the HPTA
staunch308
New member
Yeah makes sense actually, cheers 0ni