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arthritis?

Jman_898

New member
Hey guys just wondering if anyone has been told by a doc that they will eventually end up with arthritis, curious if anyone has managed to keep it at bay... i suffer some tendon damage in my left hand and have been told i will end up with arthritis, in cold weather it gives me slight pain sometimes but most of the time its alright... i try to keep it as warm as i can in the cooler months like now... are there any other usefull tips anyone might have? cheers
 
Arthritis = inflamation= oxidative stress..

Tumerica epa/dha will be your friend.. I have some books on the matter i can lend to you if you are interested. ..

Peoples cured from diet changes...
 
Stop peddling shit you know do not know about properly noobs. Arthritis due to injury can not be cured due to diet it is an degeneration of the joint due to structural damage.
 
Stop peddling shit you know do not know about properly noobs. Arthritis due to injury can not be cured due to diet it is an degeneration of the joint due to structural damage.

Dave your ****ing seriously pissing me off tonight...

Do some reading and some research.

Wheres your PHD? Nope not 1 ok ill listen to someone who has 1 in that field then...

Offer still stand Jer for the books.

How does something degrade in the first place? If it is injured there will be inflamation and oxidative stress...

You cant shoot down my attempts to help someone because you are not familure with the work i am reffering too..
 
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hmm at this stage its not arthritis, but its onset. i can understand both points but seeing as its not actualy arthritis, then theres still a chance of cure, if not atleast some form of prevention... thanks for the info noobs, please calm down guys, honestly all the arguments on this forum lately... Its not worth the Cortisol guys!
 
He has had a serious injury by the sounds of it not usually joint degeneration. It is not curable, while I have no problem with using nutrition to help the issue saying that it is curable (or leading someone to believe this without actually stating it) is wrong and if you were actually practicing could lead you to a malpractice suit. Do you know the mechanisms of arthritic change due to structural damage?
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He has had a serious injury by the sounds of it not usually joint degeneration. It is not curable, while I have no problem with using nutrition to help the issue saying that it is curable (or leading someone to believe this without actually stating it) is wrong and if you were actually practicing could lead you to a malpractice suit. Do you know the mechanisms of arthritic change due to structural damage?
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Yes i do, i also believe diet can cure cancer and many other disease! Oh sorry i mean only spontanious remission can cure then.. The diet had nothing to do with it cough wink..

Of course i cant claim to cure anything. Because we all know only drugs can cure or prevent a disease...
 
Did I say drugs no, what I suggest is an exercise program designed to promote cartilage growth and not destruction combined with nutrition that helps this. Drugs are a last resort when everything is already screwed up. When you suggest he can cure it (and you did by you statement) with just nutrition that is going beyond what can be promised especially by a first year tafe student. Giving good advice is fine but you do not have any a year of study in the basics of your course to give any credence towards a 'nutrition will cure it' ideal.

Jman what was your injury and how did it happen? Joint location? Also way is the pain like?
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Did I say drugs no, what I suggest is an exercise program designed to promote cartilage growth and not destruction combined with nutrition that helps this. Drugs are a last resort when everything is already screwed up. When you suggest he can cure it (and you did by you statement) with just nutrition that is going beyond what can be promised especially by a first year tafe student. Giving good advice is fine but you do not have any a year of study in the basics of your course to give any credence towards a 'nutrition will cure it' ideal.

Jman what was your injury and how did it happen? Joint location? Also way is the pain like?
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Actually i dont go to tafe, and no nothing i ever speak of here has anything to do with that "piece of paper course" ...

I did not ever claim i am the magical who can heal all i simply stated someone elses work who has i dno 40years of expeirence in healing what other people couldnt...

So stop picking on me and my "first year tafe course" because all im trying to do is help. + im withdrawing from effexor wich is making me want to head butt the computer with every retarded comment someone makes on this forum.

SO lets all love each other andhelp each other not take pussy shots at each other like a bunch of bitch's.
 
You just called my comments retarded and then go and say let's all just get along? That's hypocritical, and that is me putting it as nicely as possible.
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You just called my comments retarded and then go and say let's all just get along? That's hypocritical, and that is me putting it as nicely as possible.
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I didnt say your comments did i? dont be a girl..

That comment of yours then was retarded and made me want to headbutt the computer though..

If you want to be analy retentive about comments you stated that i cant claim to heal him with my 1st year tafe crap. However you missed the point i stated about "READ THE BOOK I JUST READ"

I understand why you got the shits your a Master of Exercise Rehabilitation Student, so what i said must go against everything that you have heard or been taught and i udnerstand that.

However if we listened to everything we were taught then soy good /protein bad weightifting is bad for you gardening is exercise... Obviously we dont know everything and not all things are 100% known about disease disorders and injury... So look outside the conventional box and find an answer.
 
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Once again you can not understand how someone is in the middle. I think diet helps but people claiming it cures arthritis are going beyond what they should claim. I think proper nutrition and supplementation can really help and I never disputed that fact, I dispute when people put up claims that it will cure the problem. I also stated that it should be used in conjunction with a exercise treatment that promotes healthy joints. Hey there may even be a drug that is great too that can be used in conjunction with these treatments. Put it all together and you have a well rounded treatment plan.
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Once again you can not understand how someone is in the middle. I think diet helps but people claiming it cures arthritis are going beyond what they should claim. I think proper nutrition and supplementation can really help and I never disputed that fact, I dispute when people put up claims that it will cure the problem. I also stated that it should be used in conjunction with a exercise treatment that promotes healthy joints. Hey there may even be a drug that is great too that can be used in conjunction with these treatments. Put it all together and you have a well rounded treatment plan.
Posted via Mobile Device

But he doesnt have arthritis... So technically it isnt a cure. Just prevention? So is that defined as a cure, im not sure... So Why are they going beyond what they should claim if they can?
 
SOME NOTES ON NIACINAMIDE THERAPY FOR ARTHRITIS
by William Kaufman, M.D., Ph.D.
(Reprinted with the kind permission of Charlotte Kaufman)
The (more frequent) 250 mg dose of niacinamide is 40 to 50 % more effective in the treatment of arthritis than the (less frequent) 500 mg. dose. As an illustration, see the reprint of my Tom Spies Memorial Lecture: Niacinamide, a Most Neglected Vitamin. This illustrative case history begins on page 17 column 2 and continues on page 18 column 2.
Do not use hard gelatin capsules containing 250 mg niacinamide because they do not deliver niacinamide as efficiently as 250mg niacinamide in thin gelatin capsules in the treatment of joint dysfunction (arthritis).
In my paper in J. Amer Geriat. Society, 1955 3:927-936 I noted that niacinamide (alone or combined with other vitamins) in a thousand patient-years of use has caused no adverse side effects.
Some brands of niacinamide on the market today contains excipients that act as preservatives, probably meant to prolong shelf life. Some patients have severe adverse reactions to these preparations while most do not experience any ill effects.
Niacinamide has un-gated entrance to the central nervous system. It has a strong affinity for the central nervous system's benzodiazepine receptors and causes a pleasant calmative effect. In addition, it improves central nervous system function in the kinds of central nervous symptom impairments noted in my 1943 book, starting on page 3.
Please keep in mind niacinamide is a systemic therapeutic agent. It measurably improves joint mobility, muscle strength, decreases fatigability. It increases maximal muscle working capacity, reduces or completely eliminates arthritic joint pain. Niacinamide heals broken strands of DNA and improves many kinds of CNS functioning.
Some joints are so injured by the arthritic process that no amount of niacinamide therapy will cause improvement in joint mobility, but it takes three months of niacinamide therapy before you can conclude this, since some joints are slow to heal.
WILLIAM KAUFMAN, PhD, MD
January 13,1998

Reprinted with permission from Saul AW. William Kaufman, B-3, and arthritis. J Orthomolecular Med, 2001. Vol. 16, No. 3, Third Quarter, 2001, p 189.
The world was still deep in the Great Depression when William Kaufman, MD, PhD, had already begun treating osteoarthritis with two to four grams of niacinamide daily. Now, over 60 years later, his pioneering work in orthomolecular medicine is receiving the recognition it so well deserves.
In a 1978 radio interview with Carlton Fredericks, Dr Kaufman told of how "I had one patient who was so severely arthritic that I could not bend his elbows enough to measure his blood pressure. He was one of my first patients. I gave him niacinamide for a week in divided doses, and then he could bend his arm. I took him off it and gave him a look-alike medicine (placebo). In a week he was back where he was before: his joints were stiff again.
"I arrived at my (megavitamin B-3 dosage) schedule by actually seeing the response of patients with varying degrees of arthritis. One cannot give a single large dose and get any really favorable results in arthritis... It is necessary to divide the doses so that the blood levels of niacinamide would be fairly uniform throughout the waking day."
Kaufman's findings were both plain and elegant. The greater the stiffness, the more frequent the doses. Severely crippled arthritic patients needed up to a total of 4,000 mg/day. Divided into 10 doses per day, in one to three months, patients could now get out of their chair, or bed. "If continued, they would be able comb their hair and be able to walk upstairs, so they would no longer be prisoners of the house. By the end of about three years’ treatment, they would be fully ambulatory, and this was even in the older age groups."

something i just found...
 
THE COMMON FORM OF JOINT DYSFUNCTION
by William Kaufman, M.D., Ph.D.
(Copyright 1949 William Kaufman. Copyright renewed 2001 Charlotte Kaufman. Reprinted with permission.)

To order a mint-condition, hardcover copy of the very rare, original 1949 edition of Dr. Kaufman’s arthritis book, The Common Form of Joint Dysfunction (194 pages plus references), please click here.
METHOD OF TREATMENT OF JOINT DYSFUNCTION (pages 20-29)
After completion of his physical examination, the patient was apprised of the normal and abnormal findings revealed by the clinical study. Where problems other than joint dysfunction existed, these were discussed, and appropriate therapeutic recommendations were made. The subject of joint dysfunction was then presented. The meaning of the numerical value of the patient's Joint Range Index was explained to him in terms of the Clinical Classification of Joint Function (see page 21), and the dynamic nature of joint dysfunction was described. The patient was told that joint dysfunction was reversible in time when appropriate therapy was taken.
All patients with joint dysfunction who elected to accept treatment were given niacinamide in suitable doses, either alone or in combination with other vitamins. When indicated the appropriate vitamins were prescribed in addition to niacinamide. The water-soluble vitamins used were never prescribed in aqueous solution, but as tablets or as dry powders in capsule form. When vitamin A was used, it was usually given in conjunction with vitamin D. Vitamin D was always given in conjunction with vitamin A; when vitamin D was administered in this study, the daily dosage rarely exceeded 6,000 U.S.P.units per 24 hours (14) (10) (38) (56) (59) (95).
Participation in the therapeutic program was entirely voluntary on the part of the patient. Some patients at the outset declined to accept treatment for their joint dysfunction. When a patient accepted therapy for his joint dysfunction, with each succeeding visit after the initial one, improvement or lack of improvement in his joint dysfunction was frankly discussed with him. No patient was chided because he was unwilling or unable to carry out the program of therapy as it was originally scheduled. Thus, because there was no “loss of face," most patients cooperated well and gave an accurate account of their deviations, if any, from the suggested therapeutic program. Some patients at the end of the first or second month of treatment, or at a later time, felt so much improved physically that they discontinued therapy for their joint dysfunction, mistakenly believing, in spite of advice to the contrary, that they were "cured," and required no further therapy or medical supervision. Some of these persons, who experienced a recurrence of their original pattern of symptoms upon premature cessation of therapy, returned subsequently for re-evaluation of their therapeutic needs. Other patients, who felt that they had not benefited from therapy for their joint dysfunction, did not continue with treatment though objectively they responded satisfactorily to adequate therapy, as shown by increasing values of the Joint Range Index on serial re-measurements.
Therapy was always individualized. In the therapeutic program introduced for the treatment of joint dysfunction, each patient served as his test object in the bio-assay of the dosage of niacinamide necessary to reverse his joint dysfunction. Therapy with niacinamide (used
alone or in combination with other vitamins) was not deemed successful unless there continuous, objective improvement, as judged by continuously increasing values of the Joint Range Index on consecutive reexaminations. (When a patient subsists on a low-protein diet, amounts of niacinamide that would ordinarily be adequate for the treatment of his joint dysfunction prove to be inadequate for satisfactory improvement. In this case, the dosage of niacinamide is continued at the same level, but the protein level of the diet is increased to adequate levels, with subsequent satisfactory improvement in the joint dysfunction.) (118) (120) (172).
The clinical classification of joint function in terms of the numerical values of the Joint Range Index is listed below:
Clinical Classification of Joint Function
Degree of Joint Dysfunction Joint Range index
No joint dysfunction 96-100
Slight joint dysfunction 86-95
Moderate joint dysfunction 71-85
Severe joint dysfunction 56 -70
Extremely severe joint dysfunction 55 or less
For each clinical grade of joint dysfunction, the initial dosage schedule of niacinamide suggested below effects in time such improvement in joint dysfunction as the writer has considered to be clinically satisfactory. (However, since April 1947, it was found that dosage schedules 50-100% greater than those recommended below (particularly in the moderate, severe and extremely severe grades of joint dysfunction) are therapeutically superior, as judged by the patient's clinical response.)
While the initial dosage may be increased as necessary during treatment, it is not decreased, even though the Joint Range Index increases in response to adequate therapy.
The vitamins were administered orally, usually in equal doses at equal intervals during the day, and, in severe and extremely severe joint dysfunction, during the night when the patient would spontaneously awaken from sleep. In slight grades of joint dysfunction, the daily continuous ingestion of 100 mg of niacinamide after meals and at bedtime sufficed for treatment (400 mg/24 hours). Usually adequate in moderate joint dysfunction was the continuous ingestion of 150 mg niacinamide administered every 3 hours for 6 daily doses (900 mg/24 hours). In extremely severe and severe grades of joint dysfunction, 100-150 mg niacinamide were prescribed every hour (1500-2250 mg/24 hours), every hour and a half (1110-1650 mg./24 hours), or every two hours (800-1200 mg/24 hours), depending on the severity of the joint dysfunction, the more frequent schedule being used in more severe cases (97) (51).
It has been found in the treatment of joint dysfunction that the manner in which the daily dosage of niacinamide is divided has an important bearing on the the therapeutic results achieved; e.g., 300 mg niacinamide given three times daily (900 mg/24 hours) is inferior in its therapeutic action to 150 mg niacinamide administered every 3 hours for 6 daily doses (900 mg/24 hours). Therefore, to define the type of therapy used, the writer routinely records the following data: (a) the number of milligrams or units administered per dose, and (b) the total number of milligrams or units administered per 24 hours.
No untoward effects or clinical signs of toxicity were noted when niacinamide (alone or in combination with other vitamins) was administered on the above dosage schedules to individuals for short or long periods of observation. Before 1943, mild hypoglycemia had been noted clinically in a few persons when niacinamide exceeded certain dosage levels (97) (135) (51) (62), but this phenomenon has not been observed since that time.
"ADEQUATE" AND "OPTIMAL” DOSAGE LEVELS OF NIACINAMIDE IN THE TREATMENT OF JOINT DYSFUNCTION
"Adequate" dosage of niacinamide is defined as that clinically safe dosage of niacinamide which, when ingested in divided doses throughout the day by a person with joint dysfunction whose ordinary diet is not inadequate in protein or calories, and whose joints are not subjected to excessive mechanical joint injury, will effect in time what the writer has considered to be a satisfactory pattern of increasing values of the Joint Range Index. The pattern of recovery from joint dysfunction in response to niacinamide therapy, and the numerical limits of increments in the value of the Joint Range Index which are considered to be satisfactory for the first month of therapy and for succeeding months, are described on page 24.
“Optimal” dosage of niacinamide is defined as that clinically safe dosage niacinamide which, when ingested in divided doses during the day by a person with joint dysfunction, would permit the most rapid recovery in joint function, as demonstrated by the largest possible increments in the values of the Joint Range Index in the shortest possible period of time. At present, the optimal dosage of niacinamide for the treatment of joint dysfunction has not been determined clinically, although it is hoped to approximate such a dosage level eventually. Since adequate dosages of niacinamide have given clinically satisfactory results without producing any untoward symptoms or signs of acute or chronic toxicity, no attempt has been made in this study to determine the optimal level of niacinamide therapy in the treatment of the various clinical grades of joint dysfunction.
However, as the higher dosage levels of niacinamide have been cautiously explored in the past 22 months, it has been found in severe and extremely severe joint dysfunction that divided doses of niacinamide totaling 4 or 5 grams (4,000-5,000 mg) per 24 hours are therapeutically superior to the lower dosage schedules which previously had been considered adequate. Even these higher dosage levels of niacinamide may not be optimal for the treatment of joint dysfunction.
The optimal dosage of niacinamide for the treatment of joint dysfunction, as well as the limit of human tolerance for niacinamide, can be established only in those medical centers equipped to provide careful clinical supervision, and to conduct such chemical, metabolic and clinical laboratory studies as would reveal the earliest signs of toxicity, should these occur with the administration of progressively higher dosage levels of niacinamide.
DESCRIPTION OF JOINT DYSFUNCTION AND ITS TREATMENT FOR THE PATIENT
Since the cooperation of the patient is a prerequisite for the successful therapy of joint dysfunction, it was found desirable and necessary before treatment of joint dysfunction was instituted to discuss with the patient his various clinical problems (including the dynamic nature of joint dysfunction, and its response to niacinamide treatment, and the dynamic nature of certain complicating syndromes, and their appropriate treatment), and the therapeutic goals. During the course of therapy, it may become necessary to review and amplify this discussion for the benefit of the patient as various clinical problems arise.
Joint dysfunction is the articular aspect of a generalized, usually slowly progressive metabolic disorder which is corrected in time by adequate niacinamide therapy. Since the retrograde changes in tissue structure and function which characterize this disorder occur insidiously over a period of years, many of its symptoms and signs are incorrectly attributed by laymen and physicians alike to the so-called "normal" aging process. But
these retrograde changes in morphology and function of bodily tissues are usually
reversible in time when adequate levels of niacinamide are supplied continuously to bodily tissues. The patient who takes continuously adequate amounts of niacinamide experiences, in addition to improvement in joint function, an improvement in his general health.
Theoretically, optimal nutrition must be continuously available to bodily tissues to ensure the best possible structure and function of tissues (104) (108). While we do not know what constitutes optimal nutrition, it has been demonstrated empirically that even persons eating a good or excellent diet according to present-day standards exhibit measurable impairment in ranges of joint movement which tends to be more severe with increasing age (see page 153). It has also been demonstrated that when such persons supplement their good or excellent diets with adequate amounts of niacinamide, there is, in time, measurable improvement in ranges of joint movement, regardless of the patients' ages. In general, the extent of recovery from joint dysfunction of any given degree of severity depends largely on the duration of adequate niacinamide therapy (see pages 187 and 188).
With the ingestion of adequate amounts of niacinamide continuously for a sufficient period of time, a patient whose ordinary diet is not inadequate in protein or calories, whose joints are not subjected to excessive mechanical trauma, will recover from joint dysfunction at the satisfactory rate of 6.0 to 12.0 Joint Range Index units, or better, in the first month of therapy, and 0.5 to 1.0 Joint Range Index unit, or better, for each month of therapy thereafter, until a Joint Range Index of 96-100 is reached. (Rarely, when a patient has one or more ankylosed joints, he may have no appreciable active or passive movement of these ankylosed joints, even after two years of adequate niacinamide therapy, although his other joints recover the full ranges of movement in response to such therapy. In such cases, the Joint Range Index cannot reach 96-100; e.g., when one wrist is ankylosed and has not shown increased movement in response to niacinamide therapy, the maximum Joint Range Index attainable is 90.9; and when both wrists are ankylosed, the maximal Joint Range Index attainable is 81.8.)
In general, the more severe and more chronic the patient's joint dysfunction, the slower is the rate of recovery in response to adequate niacinamide therapy, and the slower his subjective appreciation of improvement. The rate of recovery for each patient must be established empirically from serial determinations of the Joint Range Index. In order to ensure a continuously satisfactory rate of recovery from joint dysfunction, the physician must re-examine the patient at intervals during the course of niacinamide therapy. Whenever a patient taking the amounts of niacinamide prescribed by the physician, and eating a good or excellent diet, fails to make satisfactory improvement in his Joint Range Index, in the absence of excessive mechanical joint injury the niacinamide schedule must be revised upward to that level which permits satisfactory improvement. Failure of the patient to take niacinamide as directed will result in failure to improve at a satisfactory rate.
When a patient has joint dysfunction associated with obvious arthritic deformities, he is told that the physician cannot predict whether or not in his case articular deformities will resolve with adequate niacinamide therapy. However, in response to adequate niacinamide therapy for a sufficient period of time, other patients have shown partial or complete resolution of their arthritic joint deformities. Some patients with arthritic deformities show resolution of some of their joint deformities, but not of others. Only careful observation of the patient's deformities on serial re-examinations will indicate whether or not his deformities are resolving in response toadequate niacinamide therapy. In most instances, the rate of resolution of the deformities will be slow, if it occurs at all.
It cannot be predicted whether or not a given joint that appears to be completely ankylosed clinically will recover any degree of movement. It has been observed many times that joints appearing to be clinically ankylosed prior to therapy tend to have partial or complete recovery of movement in response to adequate niacinamide therapy, although some ankylosed joints have not shown any degree of movement as a result of therapy during an observation period of several years. In response to adequate niacinamide therapy over a sufficient period of time some patients have partial or complete recovery of movement in some of their ankylosed joints, but not in others. Only careful observation of the ranges of joint movement on serial re-examinations will demonstrate whether or not a given ankylosed joint can recover any degree of movement in response to adequate niacinamide therapy.
In general, in the absence of complicating factors, the higher the patient's Joint Range Index rises in response to adequate niacinamide therapy, the fewer articular symptoms he will have; and the better he will feel. However, even though the Joint Range Index increases satisfactorily in response to adequate niacinamide therapy, the patient may not feel well because of complicating syndromes which are not on the basis of aniacinamidosis. Careful clinical study is necessary in order to establish the etiology of whatever complicating syndromes may be present and, with appropriate therapy, the patient is likely to become free from articular symptoms and to feel well. However, at any time symptoms of bodily discomfort may recur which must be studied and given appropriate treatment as promptly as possible, if the patient is to feel well again. While the patient may obtain temporary relief from articular and other symptoms through the use of analgesics, narcotics, sedatives, antihistaminics and local anesthetics, only adequate treatment of joint dysfunction and the complicating syndromes is likely to give more lasting benefits.
In order to assess the effects of niacinamide therapy on joint dysfunction and on the patient's general status, the patient is usually re-studied one month after continuous niacinamide therapy has been instituted. If good progress in recovery from joint dysfunction is noted at that time, he is reexamined in two months, and thereafter every three to six months. For the most part, this schedule of re-examination is found to be satisfactory for the supervision of the therapeutic program of patients presenting the chronic problems of joint dysfunction, although when the individual's problems are of unusual complexity, or when intercurrent problems arise, the time interval between visits is shortened.
When a patient with joint dysfunction fails to make the anticipated progress in response to niacinamide therapy, he is asked if he has taken the medication as prescribed; if not, he is urged to do so. (When a patient has taken multiple vitamin capsules as prescribed and has not made satisfactory improvement in his Joint Range Index in response to such therapy, the druggist is asked how the vitamin powders were compounded. The clinical effectiveness of niacinamide seems to be lessened when niacinamide is mixed with ascorbic acid by vigorous trituration, since this favors inter-molecular reactions between niacinamide and ascorbic acid in the dry powder state. The occurrence of such inter-molecular reactions between niacinamide and ascorbic acid is hindered by the preliminary admixture of each dry powder separately with a small amount of calcium stearate (0.2%) before the final admixture by sieving.)
It is always emphasized that the patient must take his medication continuously as prescribed until such time as the supervising physician may decide, on the basis of objective clinical evidence, that it is necessary to increase the level of niacinamide therapy in order to produce continuously satisfactory improvement in the Joint Range Index.
However, certain factors other than the ingestion of inadequate amounts of niacinamide may tend to depress the Joint Range Index. These include (a) transient or persistent mechanical joint injury resulting from unusual or physical exertion (see page 79) or from psychogenically sustained hypertonia of somatic muscle (see page 115), (b) rapid and excessive gain in weight to obesity levels, (c) excessive ingestion of alcohol, (d) inadequate dietary protein. When any of these factors is operative, it is of limited value to increase the amounts of niacinamide taken by the patient in an effort to effect satisfactory improvement in the Joint Range Index. Instead, treatment should be directed toward lessening the degree of mechanical joint injury, reducing the patient's weight to the normal range, interdicting alcohol, and increasing the protein intake to adequate levels, respectively.
When indicated, the physician describes for the patient four complicating syndromes frequently coexisting with joint dysfunction, and their treatment (see page 76). Most of the articular and non-articular symptoms of a patient with joint dysfunction which are not corrected by niacininide therapy usually originate as part of these four complicating syndromes. When the patient understands the etiologic basis of his symptoms, he will not have anxiety concerning the meaning of symptoms which would otherwise seem mysterious and alarming. The patient with joint dysfunction who has one or more of these complicating syndromes is told that he will not feel well unless joint dysfunction and these coexisting syndromes are correctly identified and successfully treated, and that in order to accomplish this, his active participation in the clinical investigation and therapeutic program is required.
TYPICAL IMPROVEMENT IN MOBILITY OF A SINGIE JOINT IN RESPONSE TO LEVELS OF NIACINAMIDE THERAPY USED PRIOR TO APRIL 1947
In serial determinations of the mobility of single joints in response to levels of niacinamide therapy used prior to April 1947, it was found that niacinamide-induced recovery of full joint mobility was an orderly process. (Since April 1947, when higher dosage schedules of niacinamide were introduced (see page 21), there has been a marked reduction in the incidence of articular pain and discomfort upon maximal passive movement of the moveable joints during various stages of recovery from joint dysfunction.)
There is described below typical improvement in joint mobility, as illustrated by several sequential stages occurring during niacinamide-induced recovery of full mobility of the metacarpophalangeal (knuckle) joint.
(Figure 14 is a schematic representation of maximal passive extension of the meta-carpophalangeal joint at four successive stages (a) (b) (c) (d), during the course of niacinamide-induced recovery of full joint mobility. The line touched by the head of the arrow in (a) (b) (c) (d) indicates the upper limit of painless extension. The shaded angle in (b) and (c) indicates the range of painful passive extension.)
Figure 14(a). On the initial examination before niacinamide therapy was instituted, the metacarpophalangeal joint of the forefinger of the right hand could be extended passively to 30% of the full range of extension for this joint. No pain or discomfort was experienced by the patient during this maneuver. The examiner noted the presence of palpatory resistance from the initiation of the movement of passive extension of this metacarpophalangeal joint, and this resistance progressively increased as the joint was extended from the range of 0% to 30% of the maximal extension; the palpatory resistance at the end of the movement was graded as firm. When at the 30% level of passive extension a small increase of force in the direction of extension caused no further extension of this joint, 30% of the full range of extension was taken as the upper limit of maximum passive extension of this metacarpophalangeal joint.
Figure 14 (b). At the end of one month of continuous, adequate niacinamide therapy, maximal passive extension of this metacarpophalangeal joint increased to 60% of the full range of extension. No pain or discomfort was experienced by the patient when the metacarpophalangeal joint was extended from 0% to 40% of the full range of extension. The patient experienced localized joint pain, often severe, as the joint was passively extended from 40% to 60% of the full range of extension. The examiner's palpatory sensation indicated that movement of the joint in passive extension was free from 0% to 40%, and that there was soft, yielding resistance which progressively increased as the finger was extended at the metacarpophalangeal joint from 40% to 60% of the full range of movement. When a further small increase of the extending force did not increase the degree of extension, 60% of the full range of extension was taken as the upper limit of passive extension of this metacarpophalangeal joint. The palpatory resistance at the end of the movement of extension was rubbery.
Figure 14 (c).After months of continuous, adequate therapy with niacinamide, maximal passive extension of the metacarpophalangeal joint reached 100%; i.e., the full range of movement. Passive extension of the metacarpophalangeal joint from 0% to 85% was without pain or discomfort; passive extension from 85% to 100% was painful. The examiner's palpatory sensation indicated that the movement of this joint was free from 0% to 85%, and that there was soft resistance, which increased progressively with increasing extension of the metacarpophalangeal joint from the level of 85% to 100%. A small additional force in the direction of extension when the 100% level was reached did not cause further extension of this joint. The palpatory resistance at the end of the full range of movement (100%) was rubbery.
Figure 14(d). With a longer period of continuous, adequate niacinamide therapy, it was possible to achieve full, free and painless extension of this metacarpophalangeal joint to the level of 100%. Slight additional palpatory force in the direction of extension with the joint fully extended did not increase the amount of movement beyond the full range of extension; i.e., the 100% level. The examiner's palpatory sensation indicated that the movement of extension was free from 0% to 100% of full extension, that the resistance met at the end of this movement was firm, and that the patient experienced no pain from this maneuver.
 
now that im the one looking for an answer to a question here, and seeing all the other disputes and babble that goes on i vow NEVER to dissagree with what anyone says on this forum again lol. its honestly not worth the cortisol :p

Thanks for your help Noobs, ill give that book you have a read when i get the time champ

Thanks Dave for your help so far too, basically its on my left hand, i mainly get the soreness in the 3rd proximal interphalangeal joint.. sometimes a bit in the metacarpophalangeal joint and across the back of my hand where the tendon runs it gets sore too.... Also the tendon that passes over the 2nd metacarpophalangeal joint kind of clicks over the top of the joint (almost like its been shortened and cannot move where its supposed to normally)

Basically the whole bunch clicks about 3 times as im closing my hand to make a tight fist, if i lightly close my hand it doesnt make a noise tho...
One click is on the 2nd meta... and the other 2 clicks feel like they are to do with the 3rd finger (middle finger)...
 
Interesting problem, hands are crap as you can not really rest them or not work them, plus they are just vitally important. You said it was a tendon injury, how is it injured and is it shortened and pulling on the joint wrong and therefore causing the problems?
 
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